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One of them. And the people answered: do whatsoever thou thinkest best. Then said the priest: let us come hither unto God. And Saul asked of God: shall I go down after the Philistines? and wilt thou deliver them into mine hands? But he answered him not at that time. Then said Saul let the people come hither out of all quarters, and know and see, in whom this sin is chanced this day: for as truly as the Lord liveth which hath saved Israel, though it be in Jonathas my son, he shall die for it. But no man answered him of all the people. Then he said unto all Israel: be ye on one side, and I and Jonathas my son will be on another. And the people said unto Saul: what thou thinkest best that do. And Saul said unto the Lord God of Israel: give perfect knowledge. And Saul and Jonathas were caught, and the people escaped free. Then said Saul: cast lots between me and Jonathas my son. And Jonathas was caught. Then Saul said to Jonathas: tell me what thou hast done. And Jonathas told him and said: I tasted a little honey upon the end of my staff that was in mine hand, and see, I must die. Then said Saul God do so and so to me, except that thou die Jonathas. But the people said unto Saul: shall Jonathas die which hath so mightily help Israel? God forbid. As truly as the Lord liveth, there shall not one hair of his head fall to the ground: for he hath wrought with God this day. And so the people delivered Jonathas, that he died not. And then Saul departed from following the Philistines. And the Philistines went to their own place. And so Saul took the kingdom over Israel, and fought against all his enemies on every side: against the Moabites: against the children of Ammon: against the Edomites: against the kings of Zobah and against the.
Thirty-six patients of the original 38 enrolled were evaluated for radiographic response Table 3 ; . The overall objective response rate for these 36 patients was 36.1% 95% CI 20.8% to 53.8% ; . This included 13 patients with a PR. No CRs were observed. The response rate for all 38 patients was 34.2% 13 of 38 ; 95% CI 19.6% to 51.4% ; . This response rate includes two patients enrolled on the study who did not undergo formal response evaluation. One of these patients had an anaphylactic reaction with the.
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Data are means SE; no. of animals in parentheses. VAS, vitamin A sufficient; VAD, vitamin A deficient; d, day; RA, retinoic acid; Lm, mean chord length; Vd, volume density. * P 0.01, VAS vs. VAD, VAD 4d RA, or VAD 12d RA. P 0.01, VAD or VAD 4d RA vs. VAS. P 0.05, VAD 12d RA vs. VAD or VAD 4d RA. AJP-Lung Cell Mol Physiol VOL.
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Church is open for people to drop in during office hours, from Thursdays noon to five, Fridays and Saturdays eight in the morning until five in the afternoon. Those who would like a tour through the church at other hours may contact Ken or Marlene Hazen 9672162 ; for an appointment. This summer we have enjoyed a time of joyful worship at the Rico Community Church. Many folks have brought cookies and flowers from their gardens. Our attendance has been holding up well 30-35 ; and you are invited to come as often as you can. Early Bird Bible Study meets at 9: 00, followed by the regular Worship Service at 10: 30. Yes, we do laugh at our Community Church. Read on and chlorothiazide.
Medicines Dropped Since 1997 Beers Criteria Independent of Diagnoses 1. Phenylbutazone Butazolidin ; 6. Metoclopramide Reglan ; with seizures or epilepsy Considering Diagnoses 7. Narcotics with bladder outflow obstruction and narcotics with constipation 2. Recently started corticosteroid therapy with diabetes 8. Desipramine Norpramin ; with insomnia 3. -Blockers with diabetes, COPD or asthma, peripheral vascular 9. All SSRIs with insomnia disease, and syncope or falls 10. -Agonists with insomnia 4. Sedative hypnotics with COPD 11. Bethanechol chloride with bladder outflow obstruction 5. Potassium supplements with gastric or duodenal ulcers Medicines Added Since 1997 Beers Criteria Independent of Diagnoses 1. Ketorolac tromethamine Toradol ; 15. Desiccated thyroid 2. Orphenadrine Norflex ; 16. Ferrous sulfate 325 mg 3. Guanethidine Ismelin ; 17. Amphetamines excluding methylpenidate and anorexics ; 4. Guanadrel Hylorel ; 18. Thioridazine Mellaril ; 5. Cyclandelate Cyclospasmol ; 19. Short-acting nifedipine Procardia and Adalat ; 6. Isoxsuprine Vasodilan ; 20. Daily fluoxetine Prozac ; 7. Nitrofurantoin Macrodantin ; 21. Stimulant laxatives may exacerbate bowel dysfunction except in presence of chronic pain requiring opiate analgesics ; 8. Doxazosin Cardura ; 22. Amiodarone Cordarone ; 9. Methyltestosterone Android, Virilon, and Testrad ; 23. NonCOX-selective NSAIDs naproxen [Naprosyn], oxaprozin, and 10. Mesoridazine Serentil ; piroxicam ; 11. Clonidine Catapres ; 24. Reserpine doses 0.25 mg d 12. Mineral oil 13. Cimetidine Tagamet ; 25. Estrogens in older women 14. Ethacrynic acid Edecrin ; Considering Diagnoses 26. Long-acting benzodiazepines: chlordiazepoxide Librium ; , 33. Decongestants with bladder outflow obstruction chlordiazepoxide-amitriptyline Limbitrol ; , 34. Calcium channel blockers with constipation clidinium-chlordiazepoxide Librax ; , diazepam Valium ; , 35. Phenylpropanolamine with hypertension quazepam Doral ; , halazepam Paxipam ; , and chlorazepate 36. Bupropion Wellbutrin ; with seizure disorder Tranxene ; with COPD, stress incontinence, depression, and falls 37. Olanzapine Zyprexa ; with obesity 27. Propanolol with COPD asthma 38. Metoclopramide Reglan ; with Parkinson disease 28. Anticholinergics with stress incontinence 39. Conventional antipsychotics with Parkinson disease 29. Tricyclic antidepressants imipramine hydrochloride, doxepine 40. Tacrine Cognex ; with Parkinson disease hydrochloride, and amitriptyline hydrochloride ; with syncope or 41. Barbiturates with cognitive impairment falls and stress incontinence 42. Antispasmodics with cognitive impairment 30. Short to intermediate and long-acting benzodiazepines with 43. Muscle relaxants with cognitive impairment syncope or falls 44. CNS stimulants with anorexia, malnutrition, 31. Clopidogrel Plavix ; with blood-clotting disorders receiving and cognitive impairment anticoagulant therapy 32. Tolterodine Detrol ; with bladder outflow obstruction Abbreviations: CNS, central nervous system; COPD, chronic obstructive pulmonary disease; COX, cyclooxygenase; NSAIDs, nonsteroidal anti-inflammatory drugs; SSRIs, selective serotonin reuptake inhibitors. * Reserpine in doses 0.25 mg was added to the list. Ditropan was modified to refer to the immediate-release formulation only and not Ditropan XL and iron supplements was modified to include only ferrous sulfate. Do not consider the long-acting dipyridamole, which has better properties than the short-acting dipyridamole in older adults except with patients with artificial heart valves.
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The compact prover is installed in the pipeline through a 3-way valve or two 2-way valves as shown in Annex 2b. Three tests are carried out for the selected number of flow rates. 6.1.2.3 Various tracer methods for calibration of the flowmeter part The accuracy of the tracer method selected shall be of a higher level than the requirement to the uncertainty of the flow meter to be calibrated. Typically, the accuracy of a tracer method is not satisfactory for calibration purposes, but is more suitable for condition monitoring or inspection purposes. The tracer method shall be one of the 2 methods as described in ISO 2975 6-7. The location of the injection point must not disturb the flow profile. The location of the detection points shall be well selected and must not disturb the flow profile. If the equipment is a commercial, clamp-on ultrasonic meter the installation and use shall follow the producers' recommendations and 6.2 and chlorpheniramine.
Observed in GH3 cells when rapid effects of TRH, such as generation of inositol 1, 4, 5-trisphosphate or elevation of cytoplasmic calcium, were measured 9, 27 ; . Antisense RNA Inhibition of the Oocyte TRH Response. To ascertain if the cloned mouse TRH-R sequence is similar to endogenous rat pituitary TRH-R mRNA, we determined whether antisense RNA transcribed in vitro from a portion of the TRH-R cDNA in pBluescript pBSmTRHR1.8 ; would inhibit TRH-R expression in oocytes injected with RNA isolated from normal rat anterior pituitary glands. Injection of RNA isolated from normal rat pituitaries led to acquisition of responses to TRH and carbachol, a muscarinic agonist Fig. 3 Upper, left trace there were no intrinsic responses to TRH or carbachol in uninjected collagenase-treated oocytes. In Fig. 3 Upper, right trace, are responses from an oocyte injected with RNA from normal rat pituitaries that had been incubated with antisense TRH-R RNA. The TRH response in the oocyte was abolished, but the carbachol response was unaffected. As shown in the compilation of data in Fig. 3 Lower, when antisense RNA was allowed to hybridize to rat pituitary RNA prior to injection, the response to TRH was inhibited by 87%, whereas the response to carbachol was not significantly inhibited. Expression in COS-1 Cells. To examine both ligand binding and response characteristics of the cloned receptor in a mammalian system, we transfected COS-1 cells transiently with the TRH-R cDNA subcloned into the eukaryotic expression vector pCDM8 that contains the cytomegalovirus promoter 22 ; pCDM8mTRHR ; . No specific binding or TRHstimulated inositol phosphate formation was detectable in untransfected COS-1 cells or cells transfected with pCDM8 alone data not shown ; . [3H]methylTRH binding and displacement by TRH and chlordiazepoxide were measured Fig. 4 Upper ; . The Ki values for TRH 10 nM ; and for chlordiazepoxide 20 , uM ; agree with those found in both oocytes injected with RNA transcribed in vitro from.
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TPAs shall be responsible for the administration of the respective PEIA Dedicated Daily Cash Investment Account and PEIA Dedicated Checking Accounts, and for the full and complete reconciliation of all deposits made and checks issued on the accounts, for as long as the accounts remain open, which shall be for at least one hundred eighty 180 ; days after the date of issuance of the last check. TPAs shall furnish the PEIA with monthly bank reconciliations for the PEIA Dedicated Cash Investment Account and PEIA Dedicated Checking Account within thirty 30 ; days after the end of each month. Payment of Administrative Expenses. Professional, Contractual and Operating Expenses. All professional, contractual and operating expenses of the PEIA shall be submitted to the PEIA on a detailed invoice. Invoices will be verified by the PEIA Fiscal Officer and approved by the Chief Financial Officer. Approved invoices will be forwarded to the State Auditor for payment. Third Party Administration Expenses. Payment of the monthly administrative fee to PEIA' Third Party Administrators will be in accordance with the terms of their s respective contracts. When payments are based, either in whole or in part, on enrollment figures, the PEIA' Eligibility Section will confirm the enrollment figures. s Payments to TPAs shall be reviewed and approved by the Chief Financial Officer. Payments to Managed Care Organizations. Payments to managed care organizations providing medical coverage to PEIA members will be made in accordance with their respective contracts. The PEIA Fiscal Officer, or other designee, will verify the enrollment data for purposes of determining the correct monthly payment. Audits Hospital. Audits of hospital claims will be performed by the TPA-C. Audits will be performed for all out-patient service claims that, in the aggregate, exceed , 000 and, for selected DRGS for in-patient claims, and for other in-patient claims that are , 000 or more. The TPA-C will review an insured' medical file and compare the file s documents with the submitted claims. When discrepancies are discovered, the TPA-C will take the necessary corrective action, to include requesting a refund from the facility or deducting the over-payment from the provider' check. The TPA-C will prepare and s deliver a report to the Director each month on the activities of the hospital audit program. Insured. Audits of claims paid to the insured will be performed by the TPA-C for charges paid of , 500 or more. When discrepancies are discovered, the TPA-C will take the necessary corrective action, to include requesting a refund from the insured, or deducting the overpayment from any checks to the insured. Pharmacy. Each year, the TPA-P will conduct an audit of at least 5% of and chlorpromazine.
2004 Multiplex PCR for the simultaneous detection of Chlamydia pneumoniae, Mycoplasma pneumoniae and Legionella pneumophila in community-acquired pneumonia Miyashita, N., Saito, A., Kohno, S., Yamaguchi, K., Watanabe, A., Oda, H., Kazuyama, Y., Matsushima, T. Respiratory Medicine 98 6 ; , pp. 542-550.
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For most patients, the purpose of insulin treatment is to return blood glucose concentrations as close to the physiological norm as reasonably possible. Subcutaneous injections cannot recreate the portal delivery or the subtle momentary adjustments of insulin secretion by the pancreatic beta-cells. Oncedaily subcutaneous injection of a long-acting insulin is occasionally sufficient in type 2 diabetic patients, usually in conjunction with oral agents, but twice-daily mixtures of isophane protamine ; intermediate-acting with regular neutral ; short-acting insulin are generally preferred. However, complex basalbolus regimens more often produce a closer approximation to the normal daily profile of circulating insulin, producing a daily blood glucose profile that shows a greater resemblance to normal figure 1 ; . Nevertheless, it is accepted that such regimens do not necessarily produce lower HbA1C values in all patients, although they should in principle manage the demands of glucose homeostasis in a more physiological manner. Insulin prescribing and management are no longer the preserve of the specialist. Insulin therapy is often initiated in primary care, and patients are empowered to make crucial decisions about day-to-day adjustments of dosage. To improve communication it would be advantageous for a simpler classification of insulins; 'basal', 'bolus' and 'biphasic' would clearly signify the manner in which they are used This approach would avoid any potential misnomer from the trade names which may only allude to an inappropriate characteristic of an insulin e.g. Actrapid is not actually 'rapidacting', it is short-acting ; . A pragmatic approach should help to demystify some of the complexities of insulin administration and nomenclature for both the prescriber and patient. The British Journal of Diabetes and Vascular Disease and chlorpropamide.
4.10.2It is recommended that both the Chlordiazepoxide : Patient Discharge Sheet and the corresponding immediate discharge letter are sent to the relevant Pharmacy to enable the Pharmacy to reconcile the quantity to be supplied. 4.11 Provision of patient Information.
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ABSTRACTS AB-4614-78 Normal and Abnormal Patterns of Cerebrovascular Reserve Tested by 133Xe Inhalation -- Meyer JS Cerebrovascular Research, Houston Veterans Administration Hospital, Houston, Texas 77211 ; , Sakai F, Naritomi H, Grant P -- Arch Neurol 35: 350-359 Jun ; 1978 * Cerebrovascular functional reserve was tested by noninvasive measurement of regional cerebral blood flow nrCBF ; at rest in quiet darkness and when repeated during standard multiple psychophysiologic activation. The test was applied to normal volunteers and patients with different neurologic disorders. The test included counting, conversation, music, and observing movements, while rCBF was measured over both cerebral hemispheres, brain stem, and cerebellum. In normal persons at rest, mean gray matter flow Fg ; values were the same for each hemisphere, Highest Fg values were observed in brain stem and both frontal regions. During activation in normal persons, there was a significant increase in Fg values over both hemispheres and in brain stem. During activation, three types of abnormal rCBF responses were seen: demented patients showed no change, patients with vascular occlusion showed little or no increase over the ischemic hemisphere, and some patients with epilepsy showed excessive increases. AB-4615-78 Electrophysiological Study of Hemiplegia. Motor Nerve Conduction Velocity, Brachial Plexus Latency, and Electromyography -- Chokroverty S Neurology Service, Veterans Administration Hospital, Hines, Illinois 60141 ; , Medina J -- Arch Neurol 35: 360-363 Jun ; 1978 * Motor nerve conduction velocities of the ulnar and common peroneal but not the median nerves were substantially reduced in the affected limbs in a series of hemiplegic patients. Slowing of conduction velocity of the common peroneal nerve was related to the reduction of skin temperature in the hemiplegic limbs. Brachial plexus latencies to biceps and deltoid muscles were longer in the affected than in the unaffected sides in five of 12 hemiplegic patients. Electromyograms EMGs ; of limb muscles showed absence of spontaneous activity in 83% of patients. Spontaneous EMG activities in 12 of the 13 patients were related to an associated subclinical neuropathy or plexopathy in the involved limbs. Entrapment and traction causing subclinical or clinical neuropathy or radiculopathy may be present in some hemiplegic patients. A B-4616-78 Treatment of Acute Central Retinal Artery Occlusion -- Younge BR Department of Ophthalmology, Mayo Clinic, Rochester, Minnesota 55901 ; , Rosenbaum TJ -- Mayo Clin Proc 53: 408-410 Jun ; 1978 and chlorzoxazone.
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At the end of treatment: Most participants were completely treated from the GI symptoms, but the incidence of such symptoms was now slightly more common among the anticonvulsant-treated patients RR 1.82; 95% CI 0.40 to 8.22; p 0.70 for between-study heterogeneity ; , a difference that again did not reach statistical significance p 0.44 ; . Quality of Sleep: Only three trials Murphy 1983; Santo 1985; Tubridy 1988 ; , including a total of 181 randomized participants, described the number of participants per group presenting sleep disturbances during treatment. At 48 hrs: The incidence of sleep disturbances was slightly more common among the patients treated with other drugs than anticonvulsants RR 0.61; 95% CI 0.21 to 1.73] ; , but this difference was not statistically significant p 0.35 ; and was characterized by significant between-study heterogeneity p 0.02 ; . Similar results were reached for the chlormethiazole versus other drug subgroup comparison RR 0.44; 95% CI 0.10 to 1.96; p 0.28; p 0.02 for between-study heterogeneity ; . At the end of treatment: Not significant differences between the compared groups RR 0.81; 95% CI 0.38 to 1.73; p 0.58; p 0.56 for between-study heterogeneity ; . The subgroup analysis of chlormethiazole versus other drug showed exactly the same results. Additional medication: Two studies Koppi 1987; Radouco-Thomas 1989 ; , including a total of 79 randomized participants, described the number of participants that required additional medication to the initial schedule of randomization. Koppi 1987 including 19 participants compared mebrobamate anticonvulsant ; versus caroverine calcium channel blocking agent ; and Radouco-Thomas 1989 including 60 participants compared tetrabamate anticonvulsant ; versus chlordiazepoxide benzodiazepine ; . No significant difference was found RR 0.99; 95% CI 0.52 to 1.90; p 0.98; p 0.66 for betweenstudy heterogeneity ; . Anticonvulsant 1 versus anticonvulsant 2: Primary efficacy outcomes: Severity of overall alcohol withdrawal syndrome: Therapeutic Success: Carbamazepine versus chlormethiazole: Two studies Lucht 2003; Lucht 2003 ; , including a total of 121 randomized participants, addressed this outcome. The data synthesis showed no significant difference between the two different anticonvulsant drugs RR 1.04; 95% CI 0.98 to 1.11; p 0.23; p 0.67 for between-study heterogeneity ; . Secondary efficacy outcomes: Side Effects: Carbamazepine versus chlormethiazole.
Ylation of RGS18 was mapped to Ser49 by MS MS analysis. This study provides a new approach for the identification of novel signaling molecules in activated platelets, providing new insights into the mechanisms of platelet activation and building the basis for the development of therapeutic agents for thrombotic diseases. Blood. 2004; 103: 2088-2095 and cholestyramine.
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The effect of cimetidine, an H2-receptor blocking agent, on the secretion of e9rTcpertech netate was studied in I I dogs. In cimetidine-treated animals, there was Increased retention of 99"Tc-pertechnetate by the gastric wall as compared with the untreated animals. The resuIts indicate the potential use of cimetidine for enhanced visualization of Meckel's diverticulum, Barrett's esophagus, and the stomach, with 9er1'cpertechnetate and chondroitin.
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Fluconazole 200 mg daily or placebo. Cisapride 20 mg four times daily was started after 7 days of fluconazole or placebo dosing. Following a single dose of fluconazole, there was a 101% increase in the cisapride AUC and a 91% increase in the cisapride Cmax. Following multiple doses of fluconazole, there was a 192% increase in the cisapride AUC and a 154% increase in the cisapride Cmax. Fluconazole significantly increased the QTc interval in subjects receiving cisapride 20 mg four times daily for 5 days. See CONTRAINDICATIONS and PRECAUTIONS. ; Midazolam: The effect of fluconazole on the pharmacokinetics and pharmacodynamics of midazolam was examined in a randomized, cross-over study in 12 volunteers. In the study, subjects ingested placebo or 400 mg fluconazole on Day 1 followed by 200 mg daily from Day 2 to Day 6. In addition, a 7.5 mg dose of midazolam was orally ingested on the first day, 0.05 mg kg was administered intravenously on the fourth day, and 7.5 mg orally on the sixth day. Fluconazole reduced the clearance of IV midazolam by 51%. On the first day of dosing, fluconazole increased the midazolam AUC and Cmax by 259% and 150%, respectively. On the sixth day of dosing, fluconazole increased the midazolam AUC and Cmax by 259% and 74%, respectively. The psychomotor effects of midazolam were significantly increased after oral administration of midazolam but not significantly affected following intravenous midazolam. A second randomized, double-dummy, placebo-controlled, cross-over study in three phases was performed to determine the effect of route of administration of fluconazole on the interaction between fluconazole and midazolam. In each phase the subjects were given oral fluconazole 400 mg and intravenous saline; oral placebo and intravenous fluconazole 400 mg; and oral placebo and IV saline. An oral dose of 7.5 mg of midazolam was ingested after fluconazole placebo. The AUC and Cmax of midazolam were significantly higher after oral than IV administration of fluconazole. Oral fluconazole increased the midazolam AUC and Cmax by 272% and 129%, respectively. IV fluconazole increased the midazolam AUC and Cmax by 244% and 79%, respectively. Both oral and IV fluconazole increased the pharmacodynamic effects of midazolam. See PRECAUTIONS. ; Azithromycin: An open-label, randomized, three-way crossover study in 18 healthy subjects assessed the effect of a single 800 mg oral dose of fluconazole on the pharmacokinetics of a single 1200 mg oral dose of azithromycin as well as the effects of azithromycin on the pharmacokinetics of fluconazole. There was no significant pharmacokinetic interaction between fluconazole and azithromycin. Microbiology Fluconazole exhibits in vitro activity against Cryptococcus neoformans and Candida spp. Fungistatic activity has also been demonstrated in normal and immunocompromised animal models for systemic and intracranial fungal infections due to Cryptococcus neoformans and for systemic infections due to Candida albicans. In common with other azole antifungal agents, most fungi show a higher apparent sensitivity to fluconazole in vivo than in vitro. Fluconazole administered orally and or intravenously was active in a variety of animal models of fungal infection using standard laboratory strains of fungi. Activity has been demonstrated against fungal infections caused by Aspergillus flavus and and chooz.
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Generalization; secobarbital-lever selection by baboon LE at 18 mg kg, compared with partial generalization; methyprylon-nonmonotonic lever selection for baboon MS, rather than an orderly function, and lever selection at 56 mg kg for baboon RF rather than partial generalization. For hexobarbital fig. 8 ; , there was lorazepam lever selection at 56 mg kg p.o. for baboon MS 30 min after dosing, but there was no lorazepam lever selection for the one baboon ML ; that had otherwise been shown to generalize. For methohexital fig. 9 ; , the conclusion of partial generalization for baboon ML with i.m. dosing would change to none. Lorazepam lever selection data also were evaluated for the Bzs. For the baboon RF ; that showed a maximum of 65% lorazepam responding in any test with diazepam, the first food pellet of the test sessions at 18 and 32 mg kg was obtained after responding exclusively on the lorazepam lever. Thus, such an assessment results in a conclusion that diazepam shared discriminative stimulus effects with lorazepam in all six baboons. Two baboons failed to generalize completely to chlordiazepoxide fig. 3 ; . For one LO ; , the first test with 32 mg kg resulted in lorazepam-lever selection; but the second resulted in selection of the ND lever overall percentages in the two tests were 56 and 59%, respectively ; . Thus, averaging the results of the tests in terms of lever selection also would have yielded a 50% choice. For the other baboon LE ; that failed to generalize to chlordiazepoxide, selection of the lorazepam lever occurred in two of the four tests with 18 mg kg p.o., which mirrors the total percentage of lorazepam lever responding in the four tests i.e., 4 45% ; . Thus, conclusions about generalization to chlordiazepoxide were not altered by use of a lever selection analysis. Even for Bzs for which full generalization was shown including lorazepam ; , there were tests in which total lorazepam-lever responding was high but the ND lever was selected initially and chlorothiazide.
Muscle cells 12 ; , evoked Fos expression in OTcontaining neurons in the SON and PVN, and increased plasma levels of OT. Chlorisondamine, a drug that lowers AP by blocking nicotinic transmission in autonomic ganglia 12 ; , also elicited a significant increase in plasma OT. In addition, the increase in plasma levels of OT elicited by chlorisondamine was attenuated by counteracting the hypotension with infusion of a vasoconstricting agent, phenylephrine, suggesting that the OT release resulted from the hypotension produced by chlorisondamine. The increase in plasma OT concentration induced by these drugs was significantly correlated with the magnitude of the hypoten and cilium.
F i r and Last, Tert U. Eoc: G. multifida, "U. Eocene" flysch, Bartonian ; , Poland Bieda 1950 ; . C o Some Aquitanian species of Heterostegina e.g., blanckenhorni ; have been suggested to belong to this genus, but they do not conform to the generic diagnosis.
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