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5. All entries become the proper- 2. "Doc, I have this nightmare about a donut eating me ." Mike Pochowski, 505th, Operations Squadron. ty of Bullseye and are subject 3. "Ahh . Starbucks. When you gotta have it, you gotta have it." to editing for minor errors. Please clarify how the regression analysis Section 5.8, pages 9 & 10, Appendix 9 ; is used to "make allowance for any differences that might exist between the relatively small sample included in the audit and the whole CLL4 population". [In the audit population the mean number of cycles per patient was * for chlorambucil, * for fludarabine + cyclophosphamide and * for fludarabine audit report Table 3 ; . In the CLL4 database at the time the audit was undertaken the mean number of cycles received across the whole population was higher: * for chlorambucil, * for fludarabine + cyclophosphamide and * for fludarabine audit report Table 25 ; . Chemotherapy and associated activities account for a good deal of the total cost and might be expected to increase with the number of cycles given. It is therefore likely that the costs in the audit understate the average cost in the whole population. We speculate that this may be related to a number of patients who had not completed therapy at the time of the audit. We estimated patient cost per cycle for each arm as plus a constant term to allow for pre-treatment costs ; using a simple regression. Per patient cost for the full population is then estimated as a function of the number of cycles received. This is intended to correct for any difference between the audit dataset and the full CLL4 population in the mean number of cycles received.].
II. M. B. Herndon, Charles Anthony in Childremi. H. Treatnsent of Coxa Vsira 1106 A. ; amid Hamada, A Thirty-two-Year Kunikata. Follow128 Fatigue Frsocture of the Femoral Neck. Scientific research is a highly competitive field. Is there room for cooperation?.

A. IF YOU KNOW THAT YOU WILL NOT QUALIFY FOR COVERAGE, OR DO NOT WANT TO APPLY FOR AN UNDERWRITTEN PLAN, CHECK THIS BOX: ISSUE THE GUARANTEED ISSUE PLAN ONLY. SINCE I HAVE CHOSEN THIS OPTION, I UNDERSTAND THAT I WILL NOT BE CONSIDERED FOR AN UNDERWRITTEN PLAN. B. IF YOU ARE APPLYING FOR BOTH GUARANTEED ISSUE AND AN UNDERWRITTEN PLAN, SELECT ONE OF THE FOLLOWING: GUARANTEED ISSUE COVERAGE AT THE EARLIEST EFFECTIVE DATE, SO THAT I COVERED DURING THE UNDERWRITING PROCESS OF THE INDIVIDUAL PLAN. I UNDERSTAND THAT IF MY APPLICATION FOR THE UNDERWRITTEN PLAN IS APPROVED, I WILL AUTOMATICALLY BE TRANSFERRED TO THE UNDERWRITTEN PLAN. IF IT IS NOT APPROVED, I WILL CONTINUE TO RECEIVE GUARANTEED ISSUE. ISSUE THE GUARANTEED ISSUE PLAN ONLY IF I NOT APPROVED FOR THE UNDERWRITTEN PLAN. I UNDERSTAND THAT I WILL NOT HAVE ANY COVERAGE UNTIL MY APPLICATION FOR THE UNDERWRITTEN PLAN IS PROCESSED AND EITHER APPROVED OR DECLINED. ; BY SIGNING THIS STATEMENT I VERIFY THAT I HAVE READ AND UNDERSTOOD THE ELIGIBILITY CONDITIONS LISTED ABOVE AND THAT ALL OF THE INFORMATION IS TRUE AND CORRECT. TODAY'S DATE REQUIRED ; SIGNATURE OF APPLICANT OR LEGAL GUARDIAN PRINT NAME.

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Amusing trial, in which a Yankee lawyer rendered a just verdict. New York, Published at the Office of the Youth's cabinet. 1841 16 p.; illus.; 14 cm. Reel: 4, No. 71 Anacreon. Selected odes of Anacreor. Cambridge, Univ. Press, Hilliard and Metcalf. 1817 Translated into English verse, from the Greek, as published in Dalzel's 'Collectanea minora.' by William Biglow.; 18 p. Reel: 2, No. 79 The Ancient banner; or, Brief sketches of persons and scenes in the early history of Friends. Philadelphia, Joseph Kite & Co., printers. 1846 18 p.; 15 cm. Reel: 4, No. 72 Anderson, Joseph, 1836-1916. Two victories; a New England idyl. New York, William M. Franklin. 1867 30 p.; 23 cm. Reel: 7, No. 104 Anderson, Leroy. Half an hour's amusement at York and Jamestown; preparatory to a narrative of LaFayette's return and reception in Virginia. Richmond. 1824 31 p. Reel: 4, No. 73 Anderson, Mary Eleanor Roberts ; , 1840-1916. Scenes in the Hawaiian islands and California. New York, American tract society. [c1865] 238 p.; front., illus.; 18 cm. Reel: 7, No. 105 Anderson, Thomas, supposed author. Superstitution detected, by a Connecticut brickmaker, to which is added The downfall of despotism by [Charles Mead]. Philadelphia. 1831 24 p.; 18 cm. Reel: 4, No. 74 Andre, John, 1751-1780. Major Andre; [two poems]. n.p. [1780] Written while he was a prisoner in the American Camp.; broadside. Reel: 44, No. 1201 Andrews, Edward Wigglesworth, d. 1825. An address before the Washington benevolent society, in Newburyport, on the 22d. of Feb., 1816. Newburyport, Published by William B. Allen & co., No. 13 Cornhill. 1816 Published by request of the society.; 15 p.; front. port. ; . Reel: 2, No. 81 Andrews, H.P. The common school exhibition, containing the May queen, and fairy queen and various other original pieces suitable for declamation and school exhibitions. Boston, Fitz and Hobbs. 1849 165 p.; 18 cm. Reel: 4, No. 75 Andros, Richard Salter Storrs, 1817-1868. Chocorua, and other sketches. Fall River, William Canfield & co. MDCCCXXXVIII 88 p. Reel: 4, No. 76 Anecetus, pseud. The walk. Boston, The author. 1859 32 p.; 22.5 cm. Reel: 7, No. 107 Angel love and other poems. Worcester, Mass., Z. Baker & co. 1855 [4], 116 p.; 17 cm. Reel: 7, No. 108 Angeline, Lady, pseud. The council of wisdom. [New York?]. [1856?] Wholly original 24 p.; 20 cm.; Poetry and prose. Reel: 7, No. 109 Anglo Ab-original sleighing song, in five canters, written after Longfellow. New Haven, T.J. Stafford, printer. 1856 Air-Highwaythere. Dedicated to the Marshals and their neighbors.; 8 p. 22 cm.; Half-title. Reel: 7, No. 110 Anketell, John, b. 1750?. Poems on several subjects; to which are added, The epistle of Yarico to Inkle. Boston, Printed and sold by William Spotswood, no. 55 Marlborough-street. 1795 1 p.l., xiii, [1], 218 p.; 14 cm.; Story, Isaac. Epistle of Yarico to Inkle, p. 205-218. Reel: 2, No. 82 The Anniversary ode of the Columbian reading society. [n.p., Duane and Son Printers]. [1806] 7 p. Reel: 2, No. 83 Anniversary ode of the Union Book Society. [Washington City, Dinmore and Cooper, Printers]. 1808 10 p., 1 l., 3 p.; An oration delivered on the anniversary of the Union book society, Washington City: 3 p. at end. Reel: 2, No. 85.

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Research and development R&D ; activities are divided selectively between these different fields. In the first two, Solvay Pharmaceuticals shall continue to invest in every aspect, from R&D through to worldwide marketing. Influenza vaccines and pancreatic enzymes will be the subject of targeted investments, including R&D and licensing agreements. Gastroenterology and women's and men's health will be a downstream activity, focused on marketing. In July 2005, Solvay Pharmaceuticals completed its acquisition of Fournier Pharma. In October it presented the new combined performance objectives for the future `Solvay Pharmaceuticals' by 2010: recurrent earnings before interest and tax REBIT ; sales in excess of 20%; annual sales growth above 7%; and improving efficiency by 300 million a year. The company is already on the road to achieving these objectives. Various significant and simultaneous changes in the pharmaceuticals environment have led to adaptations within its business. Among these changes we would mention: un-met medical needs; risk benefit ratios and safety and compliance requirements that are becoming important criteria in gaining approvals; and significantly rising costs of development. Postregistration clinical work and the need to police and fluoride.
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In the context of the new conditioning approaches ranging from very light protocols "mini-transplantation" ; to those with only attenuated-intensity combination chemotherapy derived from existing conventional transplantation protocols, the treosulfan and fludarabine regimen has to be considered a more intensive conditioning approach.41 Application of almost two thirds of the maximum tolerated dose 47 g m2 after single injection11, 12, 42 ; over 3 days obviously induces sufficient myeloablation stem cell toxicity. In combination with fludarabine, potent pretransplantation immunosuppression is given to guarantee prompt engraftment and complete donor chimerism in nearly all patients. At the same time, the toxicity of this conditioning regimen has to be considered to be comparably low and the dreaded side effects of busulfan- or melphalan-based conditioning regimens such as VOD and pulmonary or cardiac toxicity have not been observed. Taking into account the low relapse rate even of patients in disease stages with high relapse risk, the conditioning with treosulfan and fludarabine may be considered a full-intensity but reduced-toxicity conditioning regimen. On the basis of the experience with other autologous or allogeneic treosulfan-containing protocols, 11, 12, 42 a study to escalate the treosulfan dose to a total of 42 g being conducted to further increase the antineoplastic potential while maintaining the low toxicity profile. Considering the suitability of combining treosulfan with, for example, cyclophosphamide, melphalan, or etoposide, 12, 42, 43 this may ultimately lead to new reduced-toxicity but full-intensity conditioning regimens. From the results reported here, promising antineoplastic activity may be anticipated in myelodysplastic syndrome, AML, multiple myeloma, and NHL. In vitro in vivo data for multiple myeloma, chronic myeloid leukemia, and acute myeloid leukemia support the potential activity of treosulfan in these indications also. This study has already shown that treosulfan and fludarabine is a favorable combination for conditioning with respect to toxicity, achievement of complete donor chimerism, and low GVHD rate and with a low treatmentrelated mortality and relapse rate. Consequently, the overall and disease-free survival rates are promising in a poor-risk patient population and fluphenazine.

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TABLE 2: Pretransplant characteristics and outcome of fludarabine refractory patients Age 50 51 56 Salvage chemotherapy CHOP Richter's transformation ; Alemtuzumab, CHOP CHOP, Alemtuzumab, Alemtuzumab Rituximab Rituximab None CHOP, Alemtuzumab, Rituximab Alemtuzumab HDMP, Alemtuzumab Rituximab Donor MR mMU mMU MR MR MR mMU MR Status at HCT PD PR PR Best response after HCT CR CR PR Outcome Relapsed shortly after HCT. Unresponsive to DLI, died of PD. Delayed graft rejection. Alive in clinical CR, but with increased proportion of CD5 CD19 + cells. Delayed graft failure. Died of RSV pneumonia after top-up transplantation. Lack of platelet engraftment. Died of PTLD after topup transplantation. Ganciclovir-induced graft failure. Alive in PR persistent thrombocytopenia ; . Relapsed 5 months post-HCT. Alive with PD. Alive in CR. Never responded to any therapy. Died of PD. Relapsed 3 months post-HCT. Complete response to DLI. Alive in CR. Died of neutropenic sepsis on day + 12 Died of a fungal cerebral abscess 3.5 months postHCT. 41. Maccubbin, A., E., Caballes, L., Riordan, J. M., Huang, D. H., and Gurtoo, H. L. A cyclophosphamide DNA phosphoester adduct formed in vitro and in vivo. Cancer Res., 51: 886 892, Den Engelse, L., De Graaf, A., De Brij, R-J., and Menkveld, G. J. O2- and O4-ethylthymine and the ethylphosphotriester dTp Et ; dT are highly persistent DNA modifications in slowly dividing tissues of the ethylnitrosourea-treated rat. Carcinogenesis Lond. ; , 8: 751757, 1987. Fortini, P., Raspaglio, G., Falchi, M., and Dogliotti, E. Analysis of DNA alkylation damage and repair in mammalian cells by the comet assay. Mutagenesis, 11: 169 175, Yamauchi, T., and Plunkett, W. UCN-01 inhibits DNA repair initiated by ultraviolet-C or by the cyclophosphamide prodrug, 4hydroperoxycyclophosphamide, and induces greater than additive cytotoxicity in normal lymphocytes. Proc. Am. Assoc. Cancer Res., 42: 557, 2001. Rao, V. A., and Plunkett, W. Mechanism of fludarabine-induced apoptosis in human lymphocytes undergoing DNA repair. Proc. Am. Assoc. Cancer Res., 42: 556, 2001. O'Brien, S. M., Kantarjian, H. M., Cortes, J., Beran, M., Koller, C. A., Giles, F. J., Lerner, S., and Keating, M. J. Results of the fludarabine and cyclophosphamide combination regimen in chronic lymphocytic leukemia. J. Clin. Oncol., 19: 1414 1420 and flurazepam. 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Figure 5. Donor chimerism results in the first 108 patients given 200 cGy TBI fludarabine before and mycophenolate mofetil cyclosporine after HLA-matched related hematopoietic stem cell transplant and flurbiprofen Rituximab is an anti-CD20 monoclonal antibody that has emerged as a useful biological agent in lymphoma. CD20 is expressed consistently in WM, thereby providing rationale for its use [29]. A small number of case series support the activity of this agent in WM. Treon et al. performed a retrospective study of 30 patients treated with up to 11 weekly doses median four ; of rituximab at 375 mg m2 week [30]. Fourteen patients 47% ; had received prior nucleoside analogs. Objective PRs were attained in eight of 30 patients 27% ; . An additional 19 patients 63% ; , however, either responded with PR 25% drop in M-protein ; or stable disease. Interestingly, despite a modest objective response rate, hematological improvements were seen in the majority: 12 of 14 86% ; anemic patients and nine of 12 75% ; thrombocytopenic patients sustained significant improvements in their blood counts. Supporting the notion that stabilization of disease may indeed be a worthy end point for WM therapy, Dimopoulos et al. reported similar findings in a prospective study of 27 patients with WM 22% previously treated with nucleoside analogs ; [31]. These patients received 4-weekly infusions of rituximab followed by a repeat 4-week course at 3 months if a PR had not yet been reached. Overall responses were seen in 44%, with an additional 37% attaining stable disease. In general, rituximab appears well tolerated and safe for use in WM. Toxicity appears similar to that reported in other diseases. As a single agent, however, response durations in WM are short median 8 months in the Treon study, 16 months in the Dimopoulos study ; and CRs elusive. Repeated dosing at regular intervals perhaps every 36 months ; may be beneficial. Rituximab's unique toxicity profile and lack of myelosuppression makes it attractive for use in WM patients either upfront or as salvage ; with severe cytopenias or poor performance status. Rituximab in combination with chemotherapy e.g. CHOP ; appears more effective than CHOP alone in aggressive histology lymphomas, and therefore warrants investigation in WM. An NCI-sponsored Eastern Cooperative Oncology Group ECOG ; study combining rituximab with fludarabine in WM is underway, and evaluations of the combination of rituximab, cyclophosphamide and a nucleoside analog are planned.

1. Kraut EH. Clinical manifestations and infectious complications of hairy-cell leukaemia. Best Pract Res Clin Haematol. 2003; 16: 33-40. Jansen J, Hermans J. Splenectomy in hairy cell leukemia: a retrospective multicenter analysis. Cancer. 1981; 47: 2066-2076. Quesada JR, Reuben J, Manning JT, Hersh EM, Gutterman JU. Alpha interferon for induction of remission in hairy-cell leukemia. N Engl J Med. 1984; 310: 15-18. Ratain MJ, Golomb HM, Vardiman JW, Vokes EE, Jacobs RH, Daly K. Treatment of hairy cell leukemia with recombinant alpha 2 interferon. Blood. 1985; 65: 644-648. Piro LD, Carrera CJ, Carson DA, Beutler E. Lasting remissions in hairy-cell leukemia induced by a single infusion of 2-chlorodeoxyadenosine. N Engl J Med. 1990; 322: 1117-1121. Estey EH, Kurzrock R, Kantarjian HM, et al. Treatment of hairy cell leukemia with 2-chlorodeoxyadenosine 2-CdA ; . Blood. 1992; 79: 882-887. Kraut EH, Bouroncle BA, Grever MR. Pentostatin in the treatment of advanced hairy cell leukemia. J Clin Oncol. 1989; 7: 168-172. Grever M, Kopecky K, Foucar MK, et al. Randomized comparison of pentostatin versus interferon alfa-2a in previously untreated patients with hairy cell leukemia: an intergroup study. J Clin Oncol. 1995; 13: 974-982. Flinn IW, Kopecky KJ, Foucar MK, et al. Longterm follow-up of remission duration, mortality, and second malignancies in hairy cell leukemia patients treated with pentostatin. Blood. 2000; 96: 2981-2986. Goodman GR, Beutler E, Saven A. Cladribine in the treatment of hairy-cell leukaemia. Best Pract Res Clin Haematol. 2003; 16: 101-116. Saven A, Burian C, Koziol JA, Piro LD. Long-term follow-up of patients with hairy cell leukemia after cladribine treatment. Blood. 1998; 92: 1918-1926. Goodman GR, Burian C, Koziol JA, Saven A. Extended follow-up of patients with hairy cell leukemia after treatment with cladribine. J Clin Oncol. 2003; 21: 891-896. Chadha P, Rademaker AW, Mendiratta P, et al. Treatment of hairy cell leukemia with 2-chlorodeoxyadenosine 2-CdA ; : long-term follow-up of the Northwestern University experience. Blood. 2005; 106: 241-246. Tallman MS, Hakimian D, Rademaker AW, et al. Relapse of hairy cell leukemia after 2-chlorodeoxyadenosine: long-term follow-up of the Northwestern University experience. Blood. 1996; 88: 1954-1959. Hoffman MA, Janson D, Rose E, Rai KR. Treatment of hairy-cell leukemia with cladribine: response, toxicity, and long-term follow-up. J Clin Oncol. 1997; 15: 1138-1142. Grever MR, Doan CA, Kraut EH. Pentostatin in the treatment of hairy-cell leukemia. Best Pract Res Clin Haematol. 2003; 16: 91-99. Dearden CE, Matutes E, Hilditch BL, Swansbury GJ, Catovsky D. Long-term follow-up of patients with hairy cell leukaemia after treatment with pentostatin or cladribine. Br J Haematol. 1999; 106: 515-519. Thomas DA, O'Brien S, Bueso-Ramos C, et al. Rituximab in relapsed or refractory hairy cell leukemia. Blood. 2003; 102: 3906-3911. Nieva J, Bethel K, Saven A. Phase 2 study of rituximab in the treatment of cladribine-failed patients with hairy cell leukemia. Blood. 2003; 102: 810-813. Hagberg H, Lundholm L. Rituximab, a chimaeric anti-CD20 monoclonal antibody, in the treatment of hairy cell leukaemia. Br J Haematol. 2001; 115: 609-611. Lauria F, Lenoci M, Annino L, et al. Efficacy of anti-CD20 monoclonal antibodies Mabthera ; in patients with progressed hairy cell leukemia. Haematologica. 2001; 86: 1046-1050. Juliusson G, Lenkei R, Liliemark J. Flow cytometry of blood and bone marrow cells from patients with hairy cell leukemia: phenotype of hairy cells and lymphocyte subsets after treatment with 2-chlorodeoxyadenosine. Blood. 1994; 83: 36723681. Ginaldi L, De Martinis M, Matutes E, Farahat N, Morilla R, Catovsky D. Levels of expression of CD19 and CD20 in chronic B cell leukaemias. J Clin Pathol. 1998; 51: 364-369. Byrd JC, Peterson BL, Morrison VA, et al. Randomized phase 2 study of fludarabine with concurrent versus sequential treatment with rituximab in symptomatic, untreated patients with B-cell chronic lymphocytic leukemia: results from Cancer and Leukemia Group B 9712 CALGB 9712 ; . Blood. 2003; 101: 6-14. Schulz H, Klein SK, Rehwald U, et al. Phase 2 study of a combined immunochemotherapy using rituximab and fludarabine in patients with chronic lymphocytic leukemia. Blood. 2002; 100: 31153120. Kanungo A, Medeiros LJ, Abruzzo LV, Lin P. Lymphoid neoplasms associated with concurrent t 14; 18 ; and 8q24 c-MYC translocation generally have a poor prognosis. Mod Pathol. 2006; 19: 25-33. Theriault C, Galoin S, Valmary S, et al. PCR analysis of immunoglobulin heavy chain IgH ; and TcR-gamma chain gene rearrangements in the diagnosis of lymphoproliferative disorders: results of a study of 525 cases. Mod Pathol. 2000; 13: 1269-1279. Campbell MJ, Zelenetz AD, Levy S, Levy R. Use of family specific leader region primers for PCR amplification of the human heavy chain variable region gene repertoire. Mol Immunol. 1992; 29: 193-203. European Bioinformatics Institute. ImMuno GeneTics IMGT ; Database. : ebi. ac imgt. Accessed October 2005. 30. Wheaton S, Tallman MS, Hakimian D, Peterson L. Minimal residual disease may predict bone marrow relapse in patients with hairy cell leukemia treated with 2-chlorodeoxyadenosine. Blood. 1996; 87: 1556-1560. Sausville JE, Salloum RG, Sorbara L, et al. Minimal residual disease detection in hairy cell leukemia. Comparison of flow cytometric immunophenotyping with clonal analysis using consensus primer polymerase chain reaction for the heavy chain gene. J Clin Pathol. 2003; 119: 213-217. Voog E, Morschhauser F, Solal-Celigny P. Neutropenia in patients treated with rituximab. N Engl J Med. 2003; 348: 2691-2694. Cervetti G, Galimberti S, Andreazzoli F, et al. Rituximab as treatment for minimal residual disease in hairy cell leukaemia. Eur J Haematol. 2004; 73: 412-417. Forconi F, Sahota SS, Raspadori D, et al. Hairy cell leukemia: at the crossroad of somatic mutation and isotype switch. Blood. 2004; 104: 33123317. Vanhentenrijk V, Tierens A, Wlodarska I, Verhoef G, Wolf-Peeters CD. V H ; gene analysis of hairy cell leukemia reveals a homogeneous mutation status and suggests its marginal zone B-cell origin. Leukemia. 2004; 18: 1729-1732. Thorselius M, Walsh SH, Thunberg U, Hagberg H, Sundstrom C, Rosenquist R. Heterogeneous somatic hypermutation status confounds the cell of origin in hairy cell leukemia. Leuk Res. 2005; 29: 153-158. Basso K, Liso A, Tiacci E, et al. Gene expression profiling of hairy cell leukemia reveals a phenotype related to memory B cells with altered expression of chemokine and adhesion receptors. J Exp Med. 2004; 199: 59-68 and fluvastatin.

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Fig. 1. Toxicity profiles of cytotoxic anticancer agents: the alkylating agents. The graphs show the IC50 log M ; for each agent against the strain panel. The vertical line is set at the IC50 of the wild-type strain. The strains are grouped and color-coded according to the DNA damage response pathway they represent. * , an accurate IC50 could not be obtained because of limiting solubility of the compound and fludarabine. Acknowledgment: The writers would like to thank Dr. Yoshikazu Kawakami Konan Hospital, Sapporo, Japan ; for his constructive comments, Miss Yoko Tani Hokkaido University School of Medicine ; for her excellent technical assistance, and Yuka Mizue Sapporo and focalin. Intensified conditioning and posttransplantation interventions to prevent relapse. Based on these considerations, we hypothesized that once-daily intravenous busulfan combined with fludarabine would yield improved safety and acceptable antileukemic activity. Here we report the results of using this combination as conditioning for HSCT in high-risk AML and MDS patients, including PK information gained using intravenous busulfan in these patients.

Floxuridine inj .FUDR .3 . fludarabine inj.FLUDARA.3 . fluorouracil inj .ADRUCIL.3 . gemcitabine inj .GEMZAR.3 . hydroxyurea caps .HYDREA .1 hydroxyurea caps .DROXIA .2 mercaptopurine tabs .PURINETHOL.1 methotrexate tabs.TREXALL.1 methotrexate inj.METHOTREXATE.3 . pemetrexed disodium inj .ALIMTA .3 . pentostatin inj .NIPENT .3 . rasburicase inj .ELITEK .3 . thioguanine tabs .THIOGUANINE TABLOID .2 ANTIMITOTICS: docetaxel inj .TAXOTERE.3 . paclitaxel inj.ABRAXANE TAXOL .3 . vinblastine inj.VELBAN .3 . DNA DEMETHYLATION AGENTS: decitabine inj.DACOGEN .3 . nelarabine inj .ARRANON.3 . IMMUNE MODULATORS AND VACCINES: aldesleukin inj.PROLEUKIN.4 . bcg vaccine live ; .TICE BCG .3 . MOLECULAR TARGET INHIBITORS: bortezomib inj .VELCADE .3 . dasatinib tab .SPRYCEL .3 erlotinib tab.TARCEVA .2 . gefitinib tab .IRESSA.3 Antineoplastics continued on next page ; Boldface indicates preferred formulary items. Brand covered with generic copayment. Requires prior approval. ! Subject to a protocol. # Quantity limits. E HIP VIP Care Improvement plan members only, Tier 5. 36 and follistim.
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The combination of fludarabine plus bexxar was well tolerated and flumist. J mol med 1999; 6-8 grever mr, lucas dm, dewald gw, et al outcome of treatment with fludarabine versus fludarabine and cyclophosphamide in chronic lymphocytic leukemia cll ; is adversely impacted by high risk genetic features: results from ecog 299 blood 2004; 1 0a abstract 3487 and formoterol.
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