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SP-P17.1: BLIND DEREVERBERATION BASED ON ESTIMATES OF SIGNAL . I - 1069 TRANSMISSION CHANNELS WITHOUT PRECISE INFORMATION OF CHANNEL ORDER Takafumi Hikichi, Marc Delcroix, Masato Miyoshi, NTT Corporation, Japan SP-P17.2: FAST ESTIMATION OF A PRECISE DEREVERBERATION FILTER BASED ON . I - 1073 SPEECH HARMONICITY Keisuke Kinoshita, Tomohiro Nakatani, Masato Miyoshi, NTT Corporation, Japan SP-P17.3: CODEBOOK-BASED BAYESIAN SPEECH ENHANCEMENT. I - 1077 Sriram Srinivasan, Jonas Samuelsson, Bastiaan Kleijn, KTH Royal Institute of Technology ; , Sweden SP-P17.4: OVERCOMING THE STATISTICAL INDEPENDENCE ASSUMPTION W.R.T I - 1081 FREQUENCY IN SPEECH ENHANCEMENT Tim Fingscheidt, Christophe Beaugeant, Suhadi Suhadi, Siemens AG, COM Mobile Phones, Germany SP-P17.5: A TWO-STAGE ALGORITHM FOR ENHANCEMENT OF REVERBERANT SPEECH . I - 1085 Mingyang Wu, Fair Isaac Corporation, United States; DeLiang Wang, The Ohio State University, United States SP-P17.6: MATRIX QUANTIZATION BASED TIME-VARYING FILTER SPEECH . I - 1089 ENHANCEMENT Sharath Rao K, Boston University, United States; Sreenivas Thippur, Indian Institute of Science, India SP-P17.7: LEAKAGE MODEL AND TEETH CLACK REMOVAL FOR AIR- AND . I - 1093 BONE-CONDUCTIVE INTEGRATED MICROPHONES Zicheng Liu, Amar Subramanya, Zhengyou Zhang, Jasha Droppo, Alex Acero, Microsoft Research, United States SP-P17.8: SPEECH ENHANCEMENT USING A MMSE SHORT TIME SPECTRAL . I - 1097 AMPLITUDE ESTIMATOR WITH LAPLACIAN SPEECH MODELING Bin Chen, Philipos Loizou, University of Texas, Dallas, United States SP-P17.9: SEPARATION OF FRICATIVES AND AFFRICATES . I - 1101 Guoning Hu, DeLiang Wang, The Ohio State University, United States SP-P17.10: SPEECH ENHANCEMENT BASED ON FILTERING THE SPECTROTEMPORAL . I - 1105 MODULATIONS Nima Mesgarani, Shihab Shamma, University of Maryland, United States SP-P17.11: IMPROVED KALMAN FILTERING FOR SPEECH ENHANCEMENT . I - 1109 Volodya Grancharov, Jonas Samuelsson, Bastiaan Kleijn, KTH Royal Institute of Technology ; , Sweden SP-P17.12: ADAPTIVE DECORRELATION FILTERING ALGORITHM FOR SPEECH SOURCE . I - 1113 SEPARATION IN UNCORRELATED NOISES Rong Hu, Yunxin Zhao, University of Missouri-Columbia, United States SP-P17.13: AN IMPROVED ESTIMATION OF A PRIORI SPEECH ABSENCE PROBABILITY . I - 1117 FOR SPEECH ENHANCEMENT : IN PERSPECTIVE OF SPEECH PERCEPTION Min Seok Choi, Hong-Goo Kang, Yonsei University, Republic of Korea.
FIG. 3. Nongenomic effects on BMD of the synthetic ligand estren. A, Model for ligand-induced dissociation of antiapoptotic from classical genomic activity of sex steroid receptors. The three diagrams depict conformational states of the receptor protein before and after interaction with the ligands, which are required to effect either the genomic genotropic ; or the antiapoptotic responses. The inactive unliganded receptor is depicted in the middle in gray. The change in conformation induced by interaction with a ligand that preferentially triggers transcriptional activity is depicted in the right in blue. The change in conformation induced by interaction with a ligand that preferentially triggers the antiapoptotic activity of the receptor e.g., the estren ; is depicted in the left in magenta. The green circle and green diamond represent the two ligands; please note the perfect and imperfect fit within the binding pocket, respectively. Ligands such as E2 will of course induce both conformations. Although in the antiapoptotic model we show direct contact between the receptor and Src, it is possible that adaptor protein s ; may bridge the interaction between the two molecules. Corresponding activation energy Ea ; of the receptor protein in the unliganded state broken line ; , progressing to either the genotropic conformation blue line ; or the antiapoptotic conformation magenta line ; , is shown at the bottom. B, Summary of the recent results presented by Kousteni et al. 96 ; and earlier studies 66, 70 ; regarding the effects of estrogens E2 ; , androgens DHT ; , and the nongenomic acting synthetic ligand estren in gonadectomized mice. Estren reproduces the nongenomic effects of sex steroids without affecting classical transcription, and it increases BMD without affecting the reproductive organs. Thus, these results indicate that its effects on BMD are exerted via nongenomic effects, whereas the reproductive effects require genomic effects. I, Increase; NC, no change. [Panel A is reproduced with permission from S. Kousteni et al.: Cell 104: 719 730, ; with permission from Elsevier Science.].
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The constant expressing the conversion of cortisol to the two tetrahydrocortisols in normal subjects was 101 f 23 Table 2 ; . This conversion was markedly decreased in both forms of the apparent mineralocorticoid excess syndrome. The differences between the two forms of the disorder were not significant, and the mean value for both forms of the syndrome was 14.1 or approximately one seventh of normal. Although the ring A reduction constant was calculated for only two patients with the type 1 variant, there is abundant evidence of decreased cortisol metabolic clearance in other type 1 patients in the form of decreased cortisol metabolite excretion 1, 22-25 ; . The clearly decreased cortisol ring A reduction constant in both forms of the syndrome of apparent mineralocorticoid excess compared to the normal value is shown in Fig. 2.
The authors wish to thank David Radtke, BS, Assistant Senior Pharmacokineticist, for his assistance with the analyses and for writing the pharmacokinetics sections of this manuscript, and Mary Alice Miller, PhD, Scientific Communications Consultant, and Noelle Gasco, Scientific Communications Associate, for their editorial assistance. This study was conducted with research support from Eli Lilly and Co.
Are the symptoms of an overactive bladder and how are they managed? A population-based prevalence study. BJU Int 87: 760-6, 2001. Stewart WF, Van Rooyen JB and Cundiff GW et al: Prevalence and burden of overactive bladder in the United States. World J Urol 20: 327-36, 2003. Hu TW and Wagner TH: Economic considerations in overactive bladder. J Manag Care, 6 Suppl 11 ; : S591-8, 2000. 8. Hu TW, Wagner TH and Bentkover JD et al: Estimated economic costs of overactive bladder in the United States. Urology 61: 1123-8, 2003. Hashim H and Abrams P: Drug treatment of overactive bladder: Efficacy, Cost and Quality-of-Life Considerations. Drugs 64: 1643-56, 2004. Andersson K-E, Appell R and Awad S et al: Pharmacological treatment of urinary incontinence, in Abrams P, Khoury S, Wein A Eds ; : Incontinence, 2nd International Consultation on Incontinence. Plymouth, Plymbridge Distributors Ltd, UK, Plymouth, 2002, pp 479-511. 11. Caulfield MP and Birdsall NJM: International Union of Pharmacology. XVII. Classification of muscarinic acetylcholine receptors. Pharmacol Rev 50: 279-90, 1998. Andersson KE: Antimuscarinics for treatment of overactive bladder. Lancet Neurol 3: 46-53, 2004. Hawthorn MH, Chapple CR and Cock M et al: Urotheliumderived inhibitory factor s ; influences on detrusor muscle contractility in vitro. Br J Pharmacol 129: 416-9, 2000. Chess-Williams RG, Manandar A and Scott RL et al: Which muscarinic receptor subtype predominates in the urothelium? Fund Clin Pharmacol 18 Suppl 1 ; : S32 abstract P03.15 ; , 2004. 15. McCloskey KD and Gurney AM: Kit positive cells in the guinea pig bladder. J Urol 168: 832-6, 2002. Gillespie JI, Harvey IJ and Drake MJ: Agonist- and nerveinduced phasic activity in the isolated whole bladder of the guinea pig: evidence for two types of bladder activity. Exp Physiol 88: 343-57, 2003. Morrison J, Steers WD and Brading A et al: Neurophysiology and neuropharmacology, in Abrams P, Khoury S, Wein A Eds ; , Incontinence, 2nd International Consultation on Incontinence. Plymouth, Plymbridge Distributors Ltd, UK, Plymouth, 2002, pp 85-161. 18. Yoshida M, Inadome A and Murakami S et al: Effects of age and muscle stretching on acetylcholine release in isolated human bladder smooth muscle. J Urol 167: 40 abstract 160 ; , 2002. 19. Andersson KE and Yoshida M. Antimuscarinics and the overactive detrusor-which is the main mechanism of action? Eur Urol 43: 1-5, 2003. Kay G, Pollack BG and Romanzi LJ. Unmasking anticholinergic load: when 1 + 1 CNS Spectr 9 12 Suppl 15 ; : S1-11, 2004. 21. Guay DR: Clinical pharmacokinetics of drugs used to treat urge incontinence. Clin Pharmacokinet 42: 1243-85, 2003. Eglen RM, Hegde SS and Watson N: Muscarinic receptor subtypes and smooth muscle function. Pharmacol Rev 48: 531-65, 1996. Beermann B, Hellstrom K and Rosen A: On the metabolism of propantheline in man. Clin Pharmacol Ther 13: 212-20, 1972. Thuroff JW, Chartier-Kastler E and Corcus J et al: Medical treatment and medical side effects in urinary incontinence in the elderly. World J Urol 16 Suppl 1 ; : S48-61, 1998. 25. Pak RW, Petrou SP and Staskin DR: Trospium chloride: a quaternary amine with unique pharmacologic properties. Curr Urol Rep 4: 436-40, 2003. Rovner ES: Trospium chloride in the management of overactive bladder. Drugs 64: 2433-46, 2004. Schladitz-Keil G, Spahn H and Mutschler E: Determination and propylthiouracil.
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| Propantheline for menSamples were obtained from 30 normal BM donors, 20 umbilical cords, and32 leukapheresis procedures. The number of CFCs and the proportion of CD34' cells in samples containing lo6 mononuclear cells from the freshly collected normalBM, cord blood, and leukapheresis productwere compared. GM-CFC numbers were significantly higher in the leukapheresis harvest median, 4, 255, range, 834 to 9, 200 ; than in normal BM median, 1, 170, range, 300 to 3, 740 ; or umbilical cord blood median, 1, 825, range, 520 to 3, 400; P .0005, Kruskal-Wallis ; . In addition, the proportion of CD34' cells was significantly higher in the ieukapheresis product median, 3.7%. range, 0.5 to 18 ; than in normal BM median, 1.48%, range, 0.4 to 8.3 ; or umbilical cord blood median, 1.13%, range, 0.56 to 3.9; P .002, Kruskal-Wallis ; . Standard long-termcultures. The behavior of hematopoietic cells from BM, umbilical cord blood, and leukapheresis product in standard long-termcultures are compared in Fig 2. Although in some cultures the numbers of supernatant CFCs increased between weeks 0 and 5, by week 8 there were fewer than week 0 in every case. The slope rate of decline ; in mean numbers of supernatant CFC did not differ significantly between the three groups P .11 ; . However, colonynumbers differed between them throughout P .0001 ; . The leukapheresis product gave rise to significantly more supernatant CFC than umbilicalcord blood P .001 ; and umbilical cord blood CFC counts were higher than that ofBM P .08 ; . These data show a highercapacityto generate CFC in the leukapheresis products than in normal BM or umbilical cord blood but give no indication of the number of stem cells. Estimation o LTCIC in leukapheresis product by limitingf dilution analysis. Data from two representative assays are shown in Fig 3 on a complementary log-log plot. Assuming that rj follows a binomial distribution, and that the number of LTCIC in any inoculum follows a Poisson distribution, the following linear relationship holds.
Combination are 1 ; both drugs have strong synergism in therapeutic efficacy CI 2 ; both drugs have strong antagonism in toxicity toward the host CI 1 3 ; the toxicities of both drugs in the host are not overlapping, and 4 ; both drugs or at least one drug ; allow significant dose reductions DRI 1 ; for a given effect. The same logic can be extended to three or more drug combinations. F. Step-by-Step Use of CompuSyn Software for Single Drug and for Drug Combination Studies Detailed procedures for using CompuSyn for automated dose-effect analysis for parameters of each drug and its combinations for quantitation simulation of synergism or antagonism are given in the users guide for CompuSyn Chou and Martin, 2005 ; . In section V., examples of applications in various fields of biomedical sciences, using old and new software, are given. In section VI. and Supplemental Data Appendices II to V, real data samples of applications using CompuSyn are shown. After data entry for doses and effects ; , the CompuSyn software generates, for a single drug, the dose-effect table, the dose-effect curve, the dose-effect parameters m, Dm, and r values ; , the median-effect plot, and the dose and effect interchange option. For multiple drugs in addition as to the single drug ; it generates the combination index table, the Fa-CI plot with simulation curves if constant ratio combinations ; , the Fa-log CI ; plot, the dose-reduction and protopic.
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Multiple sites 805.08 open 805.18 open 805.10 coccyx closed ; 805.6 with spinal cord injury closed ; 806.60 cauda equina injury 806.62 complete lesion 806.61 open 806.71 open 806.72 open 806.70 specified type NEC 806.69 open 806.79 open 805.7 collapsed 733.13 compression, not due to trauma 733.13 dorsal closed ; 805.2 with spinal cord injury - see Fracture, vertebra, with spinal cord injury, dorsal open 805.3 dorsolumbar closed ; 805.2 with spinal cord injury - see Fracture, vertebra, with spinal cord injury, dorsal open 805.3 due to osteoporosis 733.13 fetus or newborn 767.4 lumbar closed ; 805.4 with spinal cord injury closed ; 806.4 open 806.5 open 805.5 nontraumatic 733.13 open NEC 805.9 pathologic any site ; 733.13 sacrum closed ; 805.6 with spinal cord injury 806.60 cauda equina injury 806.62 complete lesion 806.61 open 806.71 open 806.72 open 806.70 specified type NEC 806.69 open 806.79 open 805.7 site unspecified closed ; 805.8 with spinal cord injury closed ; 806.8 open 806.9 open 805.9 stress any site ; 733.95 thoracic closed ; 805.2 with spinal cord injury - see Fracture, vertebra, with spinal cord injury, thoracic open 805.3 vertex - see Fracture, skull, vault vomer bone ; 802.0 open 802.1 Wagstaffe's - see Fracture, ankle wrist closed ; 814.00 open 814.10 pathologic 733.12 xiphoid process ; - see Fracture, sternum
| Address: 1Hpital L'Archet, Nice, France, 2Ventana Clinical Research, Toronto, Ontario, Canada, 3St George Dermatology, Kogarah, Australia, 4Serono International S.A., Geneva, Switzerland and 5A complete list of the principal investigators can be found at the end of this article Email: Jean-Paul Ortonne - ortonne unice ; Neil Shear - Neil.Shear sw ; Stephen Shumack - sshumack bigpond ; Eric Henninger * - eric.henninger serono * Corresponding author and protriptyline.
HE SECRETION of ACTH is controlled by several stimulatory hypothalamic hormones, including corticotropin-releasing hormone CRH ; and arginine vasopressin AVP ; 11, and possibly by an unidentified hypothalamic inhibitory factor 2, 3 ; . Endogenous opioid peptides are also involved in the basal regulation of the hypothalamo-pituitary-adrenal HPA ; axis, as administration of opioid antagonists increases peripheral ACTH or glucocorticoid concentrations in several species [human 4 ; , horse 51, and rat 611. Although the opioids are thought to act primarily at the hypothalamus 31, their effects on the various ACTH secretagogues are controversial, as reviewed in Discussion. Complicating interpretation of results is the fact that ACTH secretagogues have been measured directly during manipulations of opioid activity only in rat portal blood 7-9 ; . Workers making these measurements have pointed out that they may provide little information on the role of endogenous opioids in regulating basal ACTH secretion, as the acute surgery and anesthesia that make rat portal blood collections possible also stimulate the HPA axis 7 ; . We have developed a nonsurgical technique for cannuReceived March 28, 1995. Address all correspondence and requests for reprints to: Dr. Susan L. Alexander, Department of Endocrinology, 2nd Floor, Riverside Block, Christchurch Public Hospital, Christchurch, New Zealand. * This work was supported by grants from the U.S. NIH DK-38322; to C.H.G.I. ; and the New Zealand Health Research Council to S.L.A.
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Cremel Sbastien, Dossot Manuel, Ehrhardt Jean-Jacques Chemistry and Spectrochemistry of Interfaces, Laboratory of Physical Chemistry and Microbiology for the Environment cremel lcpme.cnrs-nancy Oxyanions and oxycations such as selenite selenate SeO32- SeO42- ; are contaminants found in ultimate nuclear wastes and several contaminated industrial soils. Batch studies have been performed mainly on powders but the scientific community need a precise understanding of the physico-chemical behavior of aqueous solutions of these contaminants towards the various crystallographic faces of soil minerals. In this presentation, contaminant adsorption will be studied on rutile TiO2 single crystals. Rutile is a well-studied oxide of environmental interest and its physico-chemical properties allow to work in a wide range of conditions pH, ionic strength, . ; [1]. As our interest is focused on the chemistry of the water oxide interface, surface specific methods are thus required. Techniques used in this work are Angle-Resolved X-Ray Photoelectron Spectroscopy, Near-Field Raman Spectroscopy NFRS ; and Surface Second Harmonic Generation SSHG ; [2]. It is shown that SSHG, which is an non-linear optical spectroscopy, is very sensitive to surface symmetry change due to molecules or ions adsorption. When contaminants are sorbed on the surface, a change of the symmetry properties and on the non-linear optical properties of the surface is detected even at low surface coverage 5 % ; . Thanks to Near-Field Raman Spectroscopy, it is possible to collect both a shear-force topography of the surface and submicronic vibrational spectra. X-Ray Photoelectron Spectroscopy was also used to study surface hydroxylation of TiO2 and to determine covering rates and chemical environnement of contaminants. [1] Dielbold, U. The surface science of titanium dioxide Surface Sci. Review 2003 48, 53229 [2] Dossot, M.; Cremel, S.; Vanderborre, J.; Grausem, J.; Humbert, B ; Drot, R.; Simoni, E.; Sorption of Uranyl Cations on a Rutile 001 ; Single Crystal Monitored by Surface SecondHarmonic Generation Langmuir 2006, 22, 140-147 and provigil.
L. Majoros * , G. Kardos, P. Feiszt and B. Szabo Department of Medical Microbiology, Medical and Health Science Center, University of Debrecen, Debrecen, Hungary.
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Definitive guidelines on therapy for the vasovagal syndrome are hampered by a dearth of randomized controlled trials, the numerous therapeutic approaches explored, and the small numbers of subjects studied. The multiple tilt protocols used at different laboratories serve to cloud the issue further. More specifically, the evaluation of therapeutic interventions in vasovagal syncope is troubled by three major methodological difficulties peculiar to this issue, namely the variable reproducibility of the head-up tilt table test, the beneficial effect of diagnosis on syncope recurrence and the tendency for vasovagal syncope to ameliorate with time.
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These fail, and if the sweating is bad enough to interfere with your work or social activities, you should ask your GP for advice. The doctor will assess whether there might be an underlying cause, and may start treatment. If necessary you may be referred to a specialist. Generalised hyperhidrosis Generalised hyperhidrosis is too widespread to treat with lotions, injections or surgery. However, some medicines taken as tablets can reduce sweating. The most reliable are those which block the chemical signal between the nerve and the sweat gland "anticholinergic" drugs such as propantheline and glycopyrrolate ; . Unfortunately, anticholinergics also affect other body functions, resulting in unwanted effects including a dry mouth, blurred vision, tummy cramps, constipation, and difficulty in passing urine. They may be harmful for people with glaucoma. A small dose is used at first and gradually increased. Some people get relief from sweating before significant side effects occur, but for many the side effects begin before they reach a dose high enough to control sweating. Localised hyperhidrosis Aluminium chloride is the usual active ingredient in commercially available antiperspirants. Stronger preparations of aluminium chloride can be prescribed for excessive sweating, and are mostly used under the arms. They should be applied twice daily for best effect. However, sore red skin is a common problem. This can be reduced by making sure the skin is completely dry before applying the solution, by using hydrocortisone cream, and by using the treatment less frequently and then trying to build up. These solutions are sometimes also useful on hands and feet. Formalin solutions harden the skin and can block the tubes leading from sweat glands to the skin surface. They are suitable only for the soles of the feet. Solutions of the anticholinergic drug glycopyrrolate can reduce sweating in localised areas. Enough may be absorbed to cause the unwanted effects mentioned above, but this is less common than with the tablets. Glycopyrrolate solutions are most likely to be effective for excessive sweating of the scalp and forehead. Iontophoresis is a method of passing a small electric current through areas of skin immersed in a dish of water, and is used mainly for the palms and soles. It was originally developed as way of getting glycopyrrolate into the skin. This can be effective, but sometimes enough is absorbed to cause side effects. The same method using only water, without any added medication, may be helpful too. Equipment for home use can be bought for a few hundred pounds. Some hospital and questran.
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