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Figure 4. A ; Detection of Gb3 formation in cultured Gla 0 and Gla 0 MAEC by autoradiography. Gla 0 and Gla 0 MAEC were grown to 85 to 90% confluence in 150-mm culture dishes. Cellular lipids were radiolabeled by addition of d[1-14C]galactose 0.2 Ci ml, specific activity; 57.0 mCi mmol ; for an additional 20 h before harvesting. Equal amounts of whole cellular lipids 400 nmol total phosphate ; were loaded directly onto HPTLC plates. Radiolabeled lipids were identified by autoradiography after separation by HPTLC. Gla 0, wild-type MAEC; Gla 0, Fabry MAEC. B ; Measurement of Gb3 levels in cultured Gla 0 and Gla 0 MAEC by HPTLC. The crude cellular lipids were extracted from either Gla 0 or Gla 0 mice at 3 to age. Neutral glycosphingolipids were purified and analyzed by HPTLC as described in Materials and Methods. The levels of Gb3 were quantified by densitometric scanning with NIH Image 1.62 software and compared with authentic Gb3 standards that consisted of 0.5, 1.0, 1.5, and 2.5 g of Gb3 lanes 1 to 5 ; run in parallel on the same plates. Lanes 6 and 7 represent the extracted glycolipids normalized for 500 nmol of total cellular phospholipids phosphate.
53. Folstein MF, Luria R. Reliability, validity and clinical application of the Visual Analogue Mood Scale. Psychol Med. 1973; 3: 479-486. Busto UE, Kaplan HL, Zawertailo LA, Sellers EM. Pharmacologic effects and abuse liability of bretazenil, diazepam, and alprazolam in humans. J Clin Pharm Ther. 1994; 55: 451-463. Versiani M, Amin M, Chouinard G. Double-blind, placebo-controlled study with reboxetine in inpatients with severe major depressive disorder. J Clin Psychopharmacol. 2000; 20: 28-34. Kathiramalainathan K, Kaplan HL, Romach MK, Busto UE, Li N-Y, Sawe J, Tyndale RF, Sellers EM. Inhibition of P450 2D6 modifies codeine abuse liability. J Clin Psychopharmacol. 2000; 20: 435-444. Ocampo JA, Busto UE, Kaplan HL, Tyndale RF, Otton SV, Nolte H, Symanzik C, Sellers EM. Does extent of p-hydroxylation alter methamphetamine kinetics? Clin Pharmacol Ther. 1996; 59: 208-212. Fabian JE, Silverstone PH. Diltiazem, a calcium chanel antagonist, partly attenuates the effects of dextroamphetamine in healthy volunteers. Int Clin Psychopharmacol. 1997; 12: 113-120. Nurnberger JI Jr, Simmons-Alling S, Kessler L, Jimerson S, Schreiber J, Hollander E, Tamminga CA, Nadi NS, Goldstein DS, Gershon ES. Separate mechanisms for behavioral, cardiovascular, and hormonal responses to dextroamphetamine in man. Psychopharmacology. 1984; 84: 200-204. Brauer LH, Ambre J, De Wit H. Acute tolerance to subjective but not cardiovascular effects of d-amphetamine in normal, healthy men. J Clin Psychopharmacol. 1996; 16: 72-76. Sofuoglu M, Brown S, Babb DA, Pentel PR, Hatsukami DK. Carvedilol affects the physiological and behavioral response to smoked cocaine in humans. Drug Alcohol Depend. 2000; 60: 69-76. Pillay SS, Renshaw PF, Bonello CM, Lafer B, Fava M, Yurgelun-Todd D. A quantitative magnetic resonance imaging study of caudate and lenticular nucleus gray matter volume in primary unipolar major depression: relationship to treatment response and clinical severity. Psychiatry Res Neuroimag. 1998; 84: 61-74. Laruelle M, Abi-Dargham A, van Dyck C, Gil R, D'Souza DC, Krystal J, Seibyl J, Baldwin R, Innis R. Dopamine and serotonin transporters in patients with schizophrenia: an imaging study with [123I] -CIT. Biol Psychiatry. 2000; 47: 371-379. Sofuoglu M, Brown S, Dudish-Poulsen S, Hatsukami DK. Individual differences in the subjective response to smoked cocaine in humans. J Drug Alcohol Abuse. 2000; 26: 591-602. De Wit H, Uhlenhuth EH, Johanson CE. The reinforcing properties of amphetamine in overweight subjects and subjects with depression. J Clin Pharm Ther. 1987; 42: 127-136. Little KY. Amphetamine, but not methylphenidate, predicts antidepressant efficacy. J Clin Psychopharmacol. 1988; 8: 177-183. Gardner EL, Ashby CR Jr. Heterogeneity of mesotelencephalic dopamine fibers: physiology and pharmacology. Neurosci Biobehav Rev. 2000; 24: 115-118. Volkow ND, Fowler JS, Wang GJ, Gur RC, Wong C, Felder C, Gatley SJ, Ding YS.
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Ackerman, M.J. ve D.E. Clapham: Ion channelsBasic sicience ano clinical disease. N. Engl. J. Med. 336: 1575, 1997. Anonim: NAcetylcysteine being investigated as "sure" for nitrate tolerance. Pharmaceut. J. 239: 586, 1987. Antman, E.M. ve di.: Nifedipine therapy for coronary arteryspasm. N. Engl. J. Med. 302: 1269, 1980. Antman, E.M. ve di.: Calcium channel blocking agents in the treatment of cardivascular disorders. Part I: Basic clinical electrophysiologic effects. Ann. Int. Med. 93875, 1980 Aronow, W.E.: Management of stable angina. N. Engl. J. Med. 289: 516, 1973. Becker, L.C.: Conditions for vasodilatorinduced coronary steal in experimental myocardial ischemia. Circulation 57: 1103, 1978. Boden, W.E. ve di.: Nifedipineinduced hypotension and myocardial ischemia in refractory angina pectoris. JAMA 253: 1311, 1985. Boden, W.E. ve di.: Incomplete Infraction Trial of European Collaborators Evaluating Prognosis Post, Thrombolysis, INTERCEPT ; : Diltiazemin acute mycardial infarction treated with thrombolytic agents: a ramdomised placebo controlled trial. Lancet. 355: 1751, 2000. Bortolotti, M. ve di.: Nifedipine in biliary and renal colic. JAMA 258: 3516, 1987. Casteello R. ve R. Hidalgo: Dipyridamoleinduced myocardial ischemia. JAMA 259: 11179, 1988. Cavero. I. ve M. Spedding: Calcium antagonists: a class of drugs with a bright future. Part I. Cellular calcium homeostasis and calcium as a coupling messenger. Life Sci. 33: 2571, 1983. Cohn, P.F.: Silent myocardial ischemia: to treat or not to treat. Hosp. Pract.: 29: 107, 1994. Corr, P.B. ve F.X. Witkowski: Arrhythmias associated with reperfusion: Basic insights and clinical relevance. J. Cardiovasc. Pharmacol. 6 Suppl. 6 ; : S 903, 1984. Cossum, P.A. ve M.S. Roberts: Metabolite inhibition of nitroglycerin metabolism in sheep tissue homogenates. J. Pharmaceut, Pharmacol. 37: 807, 1985. De Graff, A.C. ve A.F. Lyon: Evaluation of dipyridamole Persantin ; . Am. Heart. J. 65: 425, 1963. Dempsey, P.J. ve T. Cooper: Pharmacology of the coronary circulation Annu. Rev. Pharmacol. 12: 99, 1972. Editorial: Nifedipine, Lancet 2: 352, 1980. Felly, J. ve di.: Thrombolytic treatment and new calcium antagonists. Brit. Med. J. 296: 705, 1988. Fitzgerald, J.D.: Effect of betaadrenoceptive antagonists on morbidity and mortality in cardiovascular disease. Postgrad. Med. J. 52: 770, 1976. Fleckenstein, A.: Specific pharmacology of calcium in myocardium, cardiac pacemakers and vascular smooth muscle. Annu. Rev. Pharmacol. Toxical. 17: 149, 1977. Flicker, M.R. ve di.: Diltiazem and hyperkalemia, JAMA 258: 1891, 1987. Friedman, H.Z. ve di.: Death with dipyridamolethallium imaging. Ann. Int. Med. 992, 1988.
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Tell your doctor or pharmacist as soon as possible if you do not feel well while you are taking MONOPRIL. MONOPRIL helps most people with high blood pressure, but it may have unwanted side effects in a few people. All medicines can have side effects. Sometimes they are serious, most of the time they are not. You may need medical treatment if you get some of the side effects. Ask your doctor or pharmacist to answer any questions you may have. Following is a list of possible side effects. Do not be alarmed by this list. You may not experience any of them. Tell your doctor or pharmacist if you notice any of the following and they worry you: * feeling lightheaded, dizzy or faint * headache * tiredness, fatigue or weakness * dry cough * feeling sick nausea ; or vomiting * upset stomach dyspepsia ; or heartburn * diarrhoea * stomach pains * muscle cramps or pains These are the more common side effects of MONOPRIL. Mostly these are mild and short lived.
Method: NAT-2001-00963 LOD LOQ: 0.01 Micrograms Instrument Detector: HIGH PRESSURE LIQUID CHROMATOGRAPHY - FLD Media: [BEL3] - 25MM - 5.0 MICRON NYLON FILTER; 3 PIECE CASSETTE Shelf Life 1 Year Flow Rate: 2.0 Liters per Minute Rec. Vol. or Time: Sufficient volume to achieve desired LOQ based on analytical sensitivity. Call Lab. Interferences: Any compound which has the same retention time under the prescribed conditions and absorbs or emits light in the spectral area of interest are potential interferences. Compatibility Indicator: None Shipping Handling: None Method: NAT-2001-00963 LOD LOQ: 1.0 Micrograms Instrument Detector: HIGH PRESSURE LIQUID CHROMATOGRAPHY - FLD Media: [SW2] - 7.0 cm GLASS FIBER FILTER WIPE Shelf Life 1 Year Flow Rate: N A Rec. Vol. or Time: Wipe 100 Square cm Interferences: Any compound which has the same retention time under the prescribed conditions and absorbs or emits light in the spectral area of interest are potential interferences. Compatibility Indicator: None Shipping Handling: None Method: NAT-2004-07861 LOD LOQ: 0.001 Micrograms Instrument Detector: HPLC - FLD, SAMPLE ENRICHMENT Media: [TFE3] - 25 MM - 5.0 MICRON TEFLON FILTER; 3 PIECE CASSETTE Shelf Life 1 Year Flow Rate: 2.0 Liters per Minute Rec. Vol. or Time: Sufficient volume to achieve desired LOQ based on analytical sensitivity. Call Lab. Interferences: Any compound which has the same retention time under the prescribed conditions and absorbs or emits light in the spectral area of interest are potential interferences. Compatibility Indicator: None Shipping Handling: Specifically request Sample Enrichment Technique and cogentin.
Of CYP2C9 and CYP2C19 genetic polymorphisms on warfarin maintenance dose and metabolic clearance. Clin Pharmacol Ther 72: 702710. Scordo MG, Spina E, Dahl M-L, Gatti G, and Perucca E 2005 ; Influence of CYP2C9, 2C19 and 2D6 genetic polymorphisms on the steady-state plasma concentrations of the enantiomers of fluoxetine and norfluoxetine. Basic Clin Pharmacol Toxicol 97: 296 301. Scordo MG, Spina E, Facciola G, Avenoso A, Johansson I, and Dahl M-L 1999 ; Cytochrome P450 2D6 genotype and steady state plasma levels of risperidone and 9-hydroxy risperidone. Psychopharmacology 147: 300 305. Scott G, Yih L, Yeh C-M, Milosavljev S, Laurent A, and Rordorf C 2004 ; Lumiracoxib: pharmacokinetics and pharmacodynamic profile when coadministered with fluconazole in healthy subjects. J Clin Pharmacol 44: 193199. Scott J, Henderson AD, and Poffenbarger PL 1979 ; Genetic control of tolbutamide metabolism in humans. Adv Exp Med Biol 119: 361370. Scott J and Poffenbarger PL 1979 ; Pharmacogenetics of tolbutamide metabolism in humans. Diabetes 28: 4151. Sekino K, Kubota T, Okada Y, Yamada Y, Yamamoto K, Horiuchi R, Kimura K, and Iga T 2003 ; Effect of the single CYP2C9 * 3 allele on pharmacokinetics and pharmacodynamics of losartan in healthy Japanese subjects. Eur J Clin Pharmacol 59: 589 592. Setiabudy R, Kusaka M, Chiba K, Darmansjah I, and Ishizaki T 1995 ; Metabolic disposition of proguanil in extensive and poor metabolisers of S-mephenytoin 4 -hydroxylation recruited from an Indonesian population. Br J Clin Pharmacol 39: 297303. Shah RR, Oates NS, Idle JR, and Smith RL 1980 ; Genetic impairment of phenformin metabolism. Lancet 1: 1147. Shah RR, Oates NS, Idle JR, Smith RL, and Lockhart JDF 1982 ; . Impaired oxidation of debrisoquine in patients with perhexiline neuropathy. Br Med J 2959. Shaheen O, Biollaz J, Koshakji RP, Wilkinson GR, and Wood AJJ 1989 ; Influence of debrisoquin phenotype on the inducibility of propranolol metabolism. Clin Pharmacol Ther 45: 439 443. Shear NH, Spielberg SP, Grant DM, Tang BK, and Kalow W 1986 ; Differences in metabolism of sulfonamides predisposing to idiosyncratic toxicity. Ann Intern Med 105: 179 184. Shepherd AMM, Ludden TM, McNay JL, and Lin M-S 1980 ; Hydralazine kinetics after single and repeated oral doses. Clin Pharmacol Ther 28: 804 811. Shih P-S and Huang J-D 2002 ; Pharmacokinetics of midazolam and 1 hydroxymidazolam in Chinese with different CYP3A5 genotypes. Drug Metab Dispos 30: 14911496. Shimada T, Yamazaki H, Mimura M, Inui Y, and Guengerich FP 1994 ; Interindividual variations in human liver cytochrome P-450 enzymes involved in the oxidation of drugs, carcinogens and toxic chemicals: studies with liver microsomes of 30 Japanese and 30 Caucasians. J Pharmacol Exp Ther 270: 414 423. Shimamoto J, Ieiri I, Urae A, Kimura M, Irie S, Kubota T, Chiba K, Ishizaki T, Otsubo K, and Higuchi H 2000 ; Lack of differences in diclofenac a substrate for CYP2C9 ; pharmacokinetics in healthy volunteers with respect to the single CYP2C9 * 3 allele. Eur J Clin Pharmacol 56: 65 68. Shimoda K, Morita S, Yokono A, Someya T, Hirokane G, Sunahara N, and Takahashi S 2000a ; CYP2D6 * 10 alleles are not the determinant of the plasma haloperidol concentrations in Asian patients. Ther Drug Monit 22: 392396. Shimoda K, Someya T, Yokono A, Morita S, Hirokane G, Takahashi S, and Okawa M 2002 ; Impact of CYP2C19 and CYP2D6 genotypes on metabolism of amitriptyline in Japanese psychiatric patients. J Clin Psychopharmacol 22: 371378. Shimoda L, Morita S, Hirokane G, Yokono A, Someya T, and Takahashi S 2000b ; Metabolism of desipramine in Japanese psychiatric patients: the impact of CYP2D6 genotype on the hydroxylation of desipramine. Pharmacol Toxicol 86: 245249. Shirai N, Furuta T, Moriyama Y, Okochi H, Kobayashi K, Takashima M, Xiao F, Kosuge K, Nakagawa K, Hanai H, et al. 2001 ; Effects of CYP2C19 genotypic differences in the metabolism of omeprazole and rabeprazole on intragastric pH. Aliment Pharmacol Ther 15: 1929 1937. Shirai N, Furuta T, Xiao F, Kajimura M, Hanai H, Ohashi K, and Ishizaki T 2002 ; Comparison of lansoprazole and famotidine for gastric acid inhibition during the daytime and night-time in difference CYP2C19 genotype groups. Aliment Pharmacol Ther 16: 837 846. Shitara Y, Hirano M, Sato H, and Sugiyama Y 2004 ; Gemfibrozil and its glucuronide inhibit the organic anion transporting polypeptide 2 OATP2 OATP1B1: SLC21A6 ; -mediated hepatic uptake and CYP2C8-mediated metabolism of cerivastatin: analysis of the mechanism of the clinically relevant drug-drug interaction between cerivastatin and gemfibrozil. J Pharmacol Exp Ther 311: 228 236. Shon J-H, Yoon Y-R, Kim K-A, Lim Y-C, Lee K-J, Park J-Y, Cha I-J, Flockhart DA, and Shin J-G 2002 ; Effects of CYP2C19 and CYP2C9 genetic polymorphisms on the disposition of and blood glucose lowering response to tolbutamide in humans. Pharmacogenetics 12: 111119. Si D, Guo Y, Zhang Y-D, Yang L, Zhou H-H, and Zhong D 2004 ; Identification of a novel variant CYP2C9 allele in Chinese. Pharmacogenetics 14: 465 469. Siddoway LA, Thompson KA, McAllister CB, Wang T, Wilkinson GR, Roden DM, and Woosley RL 1987 ; Polymorphism of propafenone metabolism and disposition in man: clinical and pharmacokinetic consequences. Circulation 75: 785791. Sim SC, Risinger C, Dahl M-L, Aklillu E, Christensen M, Bertilsson L, and Ingelman-Sundberg M 2006 ; A common novel CYP2C19 gene variant causes ultrarapid drug metabolism relevant for the drug response to proton pump inhibitors and antidepressants. Clin Pharmacol Ther 79: 103113. 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Smallpox vaccination.37 Adverse effects of cidofovir include nephrotoxicity, neutropenia, rashes, and gastrointestinal intolerance.38 Hydroxymethylene diphosphonate cidofovir is the lipid prodrug of cidofovir that can be administered orally.39 The therapeutic management of a patient with PE should be similar to that presently used for parainfectious encephalomyelitis. The clinical presentation, neuropathologic abnormalities, and neuroimaging findings are similar between PE and parainfectious encephalomyelitis. Several studies40-42 suggest the efficacy of treatment with intravenous corticosteroid therapy in these patients. Methylprednisolone may be given in a dosage of 1 g intravenously for 3 to 5 days.43 Alternatively, intravenous methylprednisolone may be given in a dosage of 1000 mg d for 5 days, followed by 500 mg d for 3 days and then 250 mg d for 3 days.44 Either course of methylprednisolone may be followed by a 4- to 8-week course of tapering dosages of oral prednisone.43 Plasmapheresis has been used in the treatment of acute disseminated encephalomyelitis, but its efficacy in PE is unknown. 45 Given the present understanding of the pathogenesis of PE and the role of T cells, plasmapheresis is not likely to be efficacious. CONCLUSIONS The diagnosis of PE is made in the patient who has been vaccinated with the smallpox vaccine 8 to 15 days before the onset of symptoms of an altered level of consciousness, movement disorders, and convulsions, with multifocal demyelinating lesions seen on magnetic resonance imaging and a cerebrospinal fluid lymphocytic pleocytosis with a normal glucose concentration. Intravenous methylprednisolone is most likely the best initial therapy. Historically, intravenous AGG was not efficacious in the treatment of PE once this complication occurred, so intravenous immunoglobulin may similarly not be effective, but this should be investigated further. Accepted for publication February 5, 2003. Author contributions: Concept, design, and drafting of the manuscript Drs Miravelle and Roos critical revision of the manuscript for important intellectual content Dr Roos ; . Reprints not available from the authors and cognex.
In the budesonide 100 g group, 107 of 317 patients had a severe asthma exacerbation and asthma was poorly controlled on 13% of days. When the groups taking budesonide 100 g.
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A 1.000 MWe FR can in principle take up 2.5 wt% MA within its fuel composition or in the reactor core ; without major influence on the reactor physics and safety characteristics. However the availability of these types of reactors has vanished for economic and safety reasons , particularly the use of liquid sodium as coolant is a safety obstacle. The MAs would have to be incorporated in targets or specific fuel pins and kept in the reactor for 15 or more years. The mass balance of such reactor concept shows that after 4500 EFPD the depletion yield of Np and amounts to ~ 60%, but the fissioned fraction is only 20-25 %. The consumption of both MAs in the targets is about 13-14 kg TWh, a value that has to be corrected for production of MAs in the fuel itself. A specifically designed FR could serve as a "storage-transmutation" reactor for a non negligible fraction of the total annual MA production ~2, 500kg GWe dedicated FR ; . A combination of.
1992; 79: 882-7. Tallman MS, Hakimian D, Variakojis D, et al. A single cycle of 2-chlorodeoxyadenosine results in complete remission in the majority of patients with hairy cell leukemia. Blood 1992; 80: 2203-9. Seymour JF, Kurzrock R, Freireich D, Estey EH. 2-chlorodeoxyadenosine induces durable remissions and prolonged suppression of CD4 + lymphocyte counts in patients with hairy cell leukemia. Blood 1994; 83: 2906-11. Mercieca J, Matutes E, Emmett E, Coles H, Catovsky D. 2-Chlorodeoxyadenosine in the treatment of hairy cell leukaemia: differences in response in patients with and without abdominal lymphadenopathy. Br J Haematol 1996; 93: 409-11. Zinzani PL, Ascani S, Piccaluga PP, Bendandi M, Pileri S, Tura S. Efficacy of rituximab in hairy cell leukemia treatment. J Clin Oncol 2000; 18: 3875-7. Hagberg H, Lundholm L. Rituximab, a chimaeric anti-CD20 monoclonal antibody, in the treatment of hairy cell leukaemia. Br J Haematol 2001; 115: 609-11. Lauria F, Lenoci M, Annino L, Raspadori D, Marotta G, Bocchia M, et al. Efficacy of and comfrey.
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